Physical activity can increase oxidative stress levels. That’s when vitamin C should come in to work as a bit of a helpful ally. It’s involvement in the metabolism of histamine, prostaglandins, and cysteinyl leukotrienes, can help cure forms of exercise-induced bronchoconstriction.
For the study, researchers conducted a meta-analysis of three studies that showed how vitamin C halved post-exercise FEV1 decline in participants who suffered from exercise-induced bronchonostriction. Five other examined studies also showed how it helped stop respiratory sysmptoms associated with oxidative stress from exercise.
A second analysis was also carried out on 12 participants who had asthma and were an average of 26, suffering from exercise-induced bronchoconostriction. The FEV1 and FEF60 levels before and after exercise were reported on vitamin C and placebo days, but the data was not thoroughly analyzed originally.
Findings revealed that in five out of the 12 participants, exercise caused a decline greater than 60 percent in FEF60 with a dramatic FEF60 decline. Fortunately, vitamin C helped increase post-exercise FEF60 in five of the participants by 50 to 150 percent. However, no mean difference between the vitamin C and placebo days was detected in the other 7 participants.